Narcolepsy is a sleep disorder that causes an urge to fall asleep suddenly during the daytime that’s almost impossible to resist. This last anywhere between a few seconds and a few minutes.

Narcolepsy effects people between the ages of 5 and 50. However, it’s most likely to appear in young adults in their late teens and early 20s. People assigned male at birth (AMAB) have a higher risk of developing narcolepsy.

How does narcolepsy affect the body?

To understand narcolepsy, it helps to know more about the way the human sleep cycle works. That cycle involves the following stages:

• Stage 1: Light sleep. This short stage begins right after we fall asleep and accounts for about 5% of our total sleep time.
• Stage 2: Deeper sleep. This stage is deeper and makes up about 45% to 50% of all the time a person spends sleeping (this number goes up as we get older).
• Stage 3: Slow wave sleep. This stage makes up about 25% of the time we spend sleeping (this number goes down with age). It’s very hard to wake someone up in stage 3 sleep, and waking up directly from it usually causes “sleep inertia,” a state of “mental fog” and slowed thinking. This is also the stage where sleepwalking or sleep talking typically happens.
• REM sleep: REM – rapid eye movement, this stage is when we dream. When a person is in REM sleep, we can see their eyes moving beneath their eyelids.

If a person doesn’t have narcolepsy, he/she typically enter stage 1 when he/she fall asleep and then move into stages 2 and 3. The person cycle between these stages and ultimately go into REM – rapid eye movement sleep and start dreaming. After the first REM – rapid eye movement cycle, he/she start a new cycle and go back into stage 1 or 2. One cycle normally takes about 90 minutes before another begins. Most people go through four or five cycles per night (assuming they get eight hours).

If a person has narcolepsy, he/she sleep cycle doesn’t happen like that. Instead, he/she will go into the REM – rapid eye movement cycle stage shortly after falling asleep. The rest of the night, he/she will sleep only in short stretches, often without going through the typical sleep cycle.

With narcolepsy, no matter how well a person sleeps at night, he/she will feel extremely sleepy during the daytime. That urge to fall asleep is usually impossible to resist, but these sleep periods are also short (about 15 to 30 minutes) during the day. Once he/she wake up, will feel rested and ready to resume whatever he/she were doing before. However, this happens several times during the day, which is why narcolepsy is so disruptive.

Symptoms of narcolepsy

here I’ll (Dr. Qaisar Ahmed) explain different types of narcolepsy symptoms:

Excessive daytime sleepiness

This symptom happens to everyone with narcolepsy. patients with narcolepsy and experts on the condition often describe these as “sleep attacks.” All patients with narcolepsy have these sleep attacks. Patients with excessive daytime sleepiness have mental cloudiness, a lack of energy and concentration, memory lapses, a depressed mood and/or extreme exhaustion.

Sudden muscle weakness (cataplexy)

It involves sudden and brief muscle weakness that’s triggered by strong emotions, like laughter, fear, surprise, stress or anger. It can occur at any time. The attacks range from a brief buckling of the knees or slackness in the jaw or drooping of eyelids to total body paralysis with collapse. Cataplexy usually lasts a few seconds to several minutes. The patient remains fully conscious during these attacks. The rate of attacks ranges from a few in a lifetime to several per day.

Sleep-related hallucinations

These happen right after falling asleep or right before waking up.  Usually, these delusional experiences are vivid and may be frightening. The hallucinations occur just before falling asleep (called hypnagogic hallucinations) or just after waking up (called hypnopompic hallucinations). For example, seeing a person or animal in the room, feeling of floating or sensations of being touched, and hearing an alarm or voices etc.

Hallucinations during sleep paralysis are very common, and they’re often vivid and extraordinarily frightening. Fortunately, sleep paralysis is usually very short-lived, lasting only a couple of minutes at most.

Sleep paralysis

During a sleep paralysis episode, person is aware of his surroundings but cannot move or speak. But he can still move his/her eyes and breathe. Many people hear or see things that aren’t there (hallucinations), making episodes even more frightening.

While falling asleep or waking up, our brain sends signals that relax muscles in our arms and legs. The result — muscle atonia — helps us remain still during rapid eye movement (REM) sleep. With sleep paralysis, patient regain awareness but can’t move. During the rapid eye movement (REM) sleep stage, person is likely to have dreams. The brain prevents muscles in our limbs from moving to protect us from acting dreams out and hurting ourselves.

Sleep paralysis happens when a person regains awareness going into or coming out of rapid eye movement (REM). Because narcolepsy is characterized by unstable wakefulness and unstable sleep, people with narcolepsy have frequent night awakenings that can be associated with sleep paralysis.

Our brain shuts down muscle control in our body to keep us from acting out our dreams, but this should end when we wake up. However, if we have sleep paralysis, our body doesn’t regain muscle control as it should. We can still breathe and move our eyes, but we can’t talk or move the rest of our body.

Disrupted nighttime sleep

This symptom is described as frequent awakening during the night.

Automatic behavior

This symptom is described as falling asleep for several seconds but continues to perform routine tasks, such as eating, talking, driving or writing, without any awareness or later memory of ever doing the task.

Automatic movements

People with narcolepsy can often fall asleep but may keep moving parts of their body like their hands.

Amnesia or forgetfulness

It’s common for people with narcolepsy to not remember what they were doing right before falling asleep.

Sudden outbursts around sleep attacks

A person with narcolepsy may suddenly speak up and say something usually words or phrases that are nonsensical or unrelated to what’s happening around them. When someone with narcolepsy does this, it might startle them back to being fully awake, most people who do this also don’t remember doing it.

Types of narcolepsies

There are two main types of narcolepsies:

• Narcolepsy type 1: This form involves cataplexy. About 20% of narcolepsy cases are type 1.
• Narcolepsy type 2: This form doesn’t involve cataplexy. The majority of narcolepsy cases — about 80% — are type 2.

Under ordinary circumstances, our brain shuts down most muscle control in our body to keep us from acting out our dreams. People with cataplexy will have sudden muscle weakness, similar to how our body blocks movements during rapid eye movement (REM) sleep.

Mild cataplexy may only affect our face and neck — such as our jaw dropping involuntarily — or just one side of our body. Severe cataplexy can make a person collapse to the ground, which can lead to injuries. These events usually last under a few minutes, but the person may not be able to move or talk at all during that time.

Cataplexy is also unusual because certain emotions cause it to happen. Positive emotions are most likely to trigger cataplexy, especially laughing, making jokes or other humor-related behavior. Surprise, fear and anger can also trigger cataplexy, but aren’t as likely to do so.

Cataplexy can take slightly different forms in children and in people whose symptoms started within the past six months. For them, cataplexy can look like sudden, uncontrollable grimacing or face-scrunching, sticking out their tongue or loss of muscle tone (making muscles feel soft and limbs “floppy”) throughout their body without an emotion-related cause.

Type 1 narcolepsy

In 1998, researchers discovered orexins, a type of chemical molecule created and used by certain neurons for communication. The neurons that use orexins are in a part of our hypothalamus, and those neurons are key to how we stay awake.

Orexin (sometimes called hypocretins) is a molecule produced by neurons that’s usually detectable in cerebrospinal fluid (CSF). However, CSF levels of orexin are very low — or undetectable — in people with narcolepsy. That means the cells that make orexin either stopped working or something destroyed them.

According to further research, the most likely reason those neurons stopped working is an autoimmune problem. That means our immune system attacked the neurons that make and use orexins, the orexins themselves or both.

About 90% to 95% of people with type 1 narcolepsy have a specific genetic mutation (the identifier for this mutation is HLA-DQB1*06:02) that affects their immune system. However, about 25% of all people also have this mutation but don’t have narcolepsy. As a result, experts rarely test for this mutation, and they aren’t certain what role it plays. There’s also some evidence that this condition runs in families, as having a first-degree relative (a parent, sibling or child) with narcolepsy puts a person at higher risk for developing it.

However, people can also develop type 1 narcolepsy after certain viral and bacterial infections, especially strains of H1N1 influenza and bacteria like those that cause strep throat. Experts suspect that’s because infections can sometimes trigger changes and malfunctions in our immune system.

Type 2 narcolepsy

While experts know much of why type 1 narcolepsy happens, that’s not the case with type 2 narcolepsy. Experts still don’t fully understand why type 2 narcolepsy happens. But they suspect it happens for similar reasons. Those include less-severe loss of the neurons that use orexin or a problem with how orexin travels in our brain.

Secondary narcolepsy

In rare cases, narcolepsy can happen because of damage to your hypothalamus. You can have this kind of damage from head injuries (such as concussions and traumatic brain injuries), strokes, brain tumors and other conditions.

Narcolepsy can also happen as a feature of unrelated conditions that a person can inherit, for example:

• Autosomal dominant cerebellar ataxia, narcolepsy and deafness (ADCADN).
• Autosomal dominant narcolepsy, type 2 diabetes and obesity.

Causes

A doctor can suspect narcolepsy based on the symptoms. However, narcolepsy shares symptoms with several other brain- and sleep-related conditions. Because of that, the only way to diagnose narcolepsy conclusively is with specialized diagnostic tests.

Before doing most of the main tests for narcolepsy, a doctor will first make sure that the person/patient is getting enough sleep. That usually involves simple tracking methods for patient’s sleep-wake patterns, such as actigraphy. This typically uses a watch-like device that patient wear on his/her wrist to track movement patterns such as when patient’s sleeping vs. when he/she up and moving around while awake.

Tests for narcolepsy

Some possible tests for diagnosing narcolepsy include:

• Sleep study (polysomnogram): A sleep study involves multiple types of sensors that track how patient sleep. A key part of a full sleep study, formally known as a polysomnogram, is that it includes electroencephalogram (EEG) sensors. Those sensors track patient’s brain waves, allowing healthcare providers to see what stage of sleep he/she is in from minute to minute.

  Patients who have this condition go into the REM stage sleep unusually fast compared to people who don’t. They’ll also have sleep broken up by periods of wakefulness, which the sleep study can also detect and record.

  Excessive daytime sleepiness is also a main symptom of sleep apnea. A sleep study can rule out sleep apnea.

• Multiple sleep latency test (MSLT): This test involves testing whether or not patient is prone to falling asleep during the daytime. This test involves timed naps that happen in a specific timeframe. This test can help determine if a person has excessive daytime sleepiness, which is a required symptom of narcolepsy. This test often happens the following day after an overnight sleep study.
• Maintenance of wakefulness test: This test determines if patient can stay awake during the daytime, even in situations where it would be easy to fall asleep. While it’s not common in testing for narcolepsy, it’s still possible and can rule out other issues. It’s also useful for testing if stimulant treatments are helping.
• Spinal tap (lumbar puncture): This test can help determine if the orexin levels in patient’s CSF are low. This is a keyway to diagnose type 1 narcolepsy. Low orexin levels can also signal that someone with narcolepsy may develop cataplexy, even if they haven’t yet shown that symptom. Unfortunately, orexin levels don’t change in people with type 2 narcolepsy, so it isn’t always a test that helps with diagnosis.

Other tests

Many other tests are also common for people who have narcolepsy. An example of why this might happen is having cataplexy as a symptom. Cataplexy is similar to several other motor (movement-related) symptoms of brain conditions, such as atonic seizures (also known as drop attacks).

Because of that, healthcare providers might first test for more severe conditions like seizures and epilepsy, which means narcolepsy may take longer for providers to pinpoint and diagnose. Other tests are possible, too, and a doctor is the best person to explain what tests they recommend and why.

With allopathic treatment, narcolepsy is treatable but not curable. Treatments usually start with medications, but changes to patient’s daily routine and lifestyle can also help. Narcolepsy is permanent once a person develops it, so with allopathic treatment it’s a lifelong condition. However, it doesn’t get worse over time.

Most allopathic medications target excessive daytime sleepiness, but some target other symptoms, too. Possible allopathic medications for narcolepsy include:

• Wakefulness medications. These are usually the first line of allopathic treatment, for example: modafinil and armodafinil. These medications stimulate nervous system, which can help reduce the severity or frequency of daytime sleepiness.
• Amphetamines and amphetamine-like stimulants. Drugs like methylphenidate or amphetamine/dextroamphetamine combinations.
• Antidepressants. Medications like serotonin-norepinephrine reuptake inhibitors (SNRIs) such as venlafaxine, selective serotonin reuptake inhibitors (SSRIs) like fluoxetine, or tricyclic antidepressants like clomipramine or protriptyline.
• Sodium oxybate. This drug can help patient sleep and also reduces how often cataplexy happens. Most countries control this drug very tightly because of its dangerously severe side effects, but it’s still used frequently for treating type 1 narcolepsy.
• Histamine-affecting drugs. An example of this type of drug is pitolisant, a histamine receptor antagonist. Receptor antagonists are drugs that block specific chemicals in our body from attaching to cells. That slows down or prevents the cells from doing certain things.

Treatment options in allopathy for children are very limited.

Complications/side effects of narcolepsy allopathic treatment

Many medications that treat narcolepsy or its symptoms are more likely to interact with other drugs. High blood pressure (hypertension) and irregular heart rhythms are just two possible complications with stimulant medications of any kind. Sodium oxybate in particular is dangerous if combined with other drugs that dampen how your central nervous system works, and you should never mix it with alcohol.

Homeopathic treatment for narcolepsy

Homoeopathy’s strength lies in its evident effectiveness as it takes a holistic approach towards the sick individual through promotion of inner balance at mental, emotional, spiritual and physical levels. When NARCOLEPSY is concerned there are many effective medicines available in Homoeopathy, selection depends upon the individuality of the patient, considering mental and physical symptoms. With Homeopathic treatment narcolepsy is completely curable in just few days.

Hera are very few medicines for narcolepsy proven best in my (Dr. Qaisar Ahmed) practice:

Calcaria Carbonica

Flabby fat patients. A jaded state, mental or physical, due to overwork. Pituitary and thyroid disfunction. Apprehensive; fears loss of reason, misfortune, contagious diseases. Forgetful, confused, low-spirited. Anxiety with palpitation. Obstinacy: slight mental effort produces hot head. Averse to work or exertion.

Vertigo on ascending, and when turning head. Headache from overlifting, from mental exertion, with nausea. Head feels hot and heavy, with pale face. Icy coldness in, and on the head, especially right side. Open fontanelles: head enlarged; much perspiration, wets the pillow. Itching of the scalp. Scratches head on waking.

Acid Phosphoricum

Mental debility. Listless. Impaired memory. Dizziness. Dullness and indolence of mind, with want of imagination. Weakness of memory. Stupefaction in forehead, with somnolence without snoring, with closed eyes.

Antimunium Crudum

The mental symptoms and those of the gastric sphere, determine its choice, excessive irritability and fretfulness, together with a thickly coated white tongue, are true guiding symptoms to many forms of disease. Strong inclination to sleep during the day, especially in the evening or morning. Sleepy and weary. Drowsiness, especially in elderly people. Deep unrefreshing sleep. Cross and contradictive; whatever is done fails to give satisfaction. Sulky; does not wish to speak. Peevish; vexed without cause. Child cannot bear to be touched or looked at. Angry at every little attention. Sentimental mood.

Antimunium Tartaricum

Trembling of whole body, great prostration and faintness. Irresistible inclination to sleep. Great drowsiness. Excessive yawning. On falling asleep electric-like shocks. Cries during sleep with fixed eyes and trembling. Jerking up of limbs during sleep. Vertigo alternates with drowsiness. Great despondency. Fear of being alone. Muttering, delirium, and stupor. Vertigo, with dullness and confusion. Band-like feeling over forehead. Face pale and sunken. Child will not be touched without whining.

Agaricus Muscarious

Aversion to conversation. Indisposed to perform any kind of labor, especially mental. Exuberant fancy. Ecstasy. Narcolepsy: falling asleep during talking, inclination to make verses and to prophesy. Mania, timid, or furious, with great display of strength. Silly merriness. Delirium tries to get out of bed. Delirium constant, knows no one, throws things. Delirium tremens. Morose, self-willed, stubborn, slow in learning to walk and talk. Dizziness.

Arsenicum Album

Drowsy and sleepiness in the daytime while sitting. Talks in sleep. Yawning and stretching of limbs. Awakened by pains. Shocks on dropping to sleep. Violent starting with twitching, trembling of limbs during sleep. Disturbed sleep at night with great restlessness. Great anguish and restlessness. Changes place continually. Fears, of death, of being left alone. Great fear, with cold sweat. Thinks it useless to take medicine. Suicidal. Hallucinations of smell and sight. Despair drives him from place to place. Miserly, malicious, selfish, lacks courage. General sensibility increased. Sensitive to disorder and confusion. Great weakness of memory. Stupidity and dullness. Delirium, with great flow of ideas. Narcolepsy: loss of consciousness, and of sensation; dotage; maniacal actions and frenzy. Madness; loss of mind.

Pulsatilla Pratensis

Sad, crying readily; weeps when talking; changeable, contradictory. Great sensitiveness. Wants the head high. Feels uncomfortable with only one pillow. Lies with hands above head. Weeps easily. Timid, irresolute. Fears in evening to be alone, dark, ghost. Likes sympathy. Children like fuss and caresses. Easily discouraged. Morbid dread of the opposite sex. Religious melancholy. Given to extremes of pleasure and pain. Highly emotional. Narcolepsy. Neuralgic pains. Headache from overwork.

Casticum

Sleepiness during the day, yawning and drowsiness, must lie down.  Yawning and stretching. Very drowsy, can hardly keep awake. Laughs and cries during sleep.

Canabies Indica

Excessive loquacity. Hallucinations and imaginations innumerable. Narcolepsy. Very absentminded. Sudden loss of memory. Great desire to lie down in the daytime. Thoroughly exhausted after a short walk. Felt so weak that he could scarcely speak, and soon fell into a deep sleep.

Nux Muscata

Narcolepsy. Irresistibly drowsy, sleepy, muddled, as if intoxicated. Great drowsiness and sleepiness. Sleepy attacks, sudden with vertigo. Great sleepiness with all complaints. Dreams of falling from high place of being pursued.

Sepia Officianalis

Indifferent to those loved best. Averse to occupation, to family. Irritable; easily offended. Dreads to be alone. Very sad. Weeps when telling symptoms. Miserly. Anxious toward evening; indolent.  Prodromal symptoms of apoplexy. Jerking of head backwards and forwards.

Gelsemium

Drowsiness and long sound sleep. Narcolepsy, sleepiness, great drowsiness, sneezing, double vision, loss of sight. Restless at night’s sleep mostly towards morning hours. Drowsiness is felt when trying to study. Irritable, sensitive; desires to be let alone. Incapacity to think or fix the attention. Vivacity, carelessness, followed by depression of spirits. Unconnected ideas. Unconsciousness. Delirium in sleep; half-waking, with incoherent talk. Acts as if crazy, brandishes a sword in a threatening manner. Stupor cannot open the eyes. Dullness of the mental faculties.

Cimex Lectularius

Great drowsiness falls asleep when sitting in the morning. Irresistible sleepiness. Frequent yawning. Frequent yawning with cold feeling on skin.

Silicia Tera

Yielding, faint-hearted, anxious. Nervous and excitable. Drowsy all time, narcolepsy. Sensitive to all impressions. Brain-fag. Obstinate, headstrong children. Abstracted. Apathy and indifference. Weakness of memory. Incapacity for reflection. Great distraction. Tendency to misapply words in speaking. Fixed ideas: the patient thinks only of pins, fears them, searches for them, and counts them carefully. Headaches from fasting. Vertigo from looking up. Despondency, melancholy, and disposition to weep. Nostalgia. Anxiety and agitation; yielding, anxious mood. Taciturnity; concentration in self. Inquietude and ill-humor on the least provocation, arising from excessive nervous debility. Scruples of conscience. Restless and fidgety; great liability to be frightened.

Cyclamen

Great inclination to lie down, and to sleep. No desire to work. Great dejection and melancholy. Memory alternately quick and weak. Dullness and confusion of mind, narcolepsy.

Hydrocyanic Acidum

Narcolepsy. Irresistible drowsiness. Yawning and shivering. Frequent yawning, sometimes with shuddering and spasm of the skin. Drowsiness, sometimes uncontrollable, or else after a meal; heavy sleep in the morning; continued inclination to sleep; coma vigil; somnolence. Wakefulness: to sleep is difficult, almost impossible; frequent awakening. Lively dreams, without connection; anxious, disquieting dreams, dreams of death.

Kalium Bromatum

Unconscious of what is occurring around. Extreme drowsiness. Drops asleep in char. Memory absolutely destroyed; anemia; emaciation. Loss of memory – amnesic aphasia (had to be told the word before he could speak it). Mentally dull, torpid; perception slow, answers slowly. Benumbed sensation of brain. Hallucinations of sight and sound, with or without mania, precede brain and paralytic symptoms. Delirium with delusions.

Kali Nitricum

Drowsiness and sleepiness in daytime. weakness and drowsiness. Narcolepsy. Disturbed sleep at night, constant dreaming, with frequent awakening. Disturbed sleep at night; constant dreaming, with frequent awakening. Coma somnolent. Anxious and uneasy dreams.

Mercurius

Narcolepsy: excessive sleepiness, day and night; deep and prolonged sleep. Great sleepiness during day. Inclination to sleep without the power to do so. Sleep retarded in evening, and too early awakening in morning. Failing asleep late; complaints preventing sleep (as toothache, or any severe pain or trouble, &c.); sleeplessness in general before midnight. Very light and unquiet sleep, with frequent awaking, starts, and fright. Sleeplessness from nervous excitability. Frequent, anxious, horrible, fantastic, historical, vivid and voluptuous dreams.

Natrum Muriaticum

Narcolepsy: great drowsiness during day, with frequent yawning. Retarded sleep, and sleeplessness at night, with ineffectual efforts to go to sleep. Difficulty in falling asleep again, at night, after awaking. Difficulty in waking, and excessively drowsy lassitude early in morning. Agitated sleep, full of vivid and lascivious dreams with tears and talking during sleep. Ebullition of blood at night, with anxious heat (perspiration, violent throbbing of the arteries) and palpitation of heart. Nightmare. Somnambulism. At night, pains in back, quivering, apparently of the nerves, frequent emission of urine, headache, colic, asthmatic sufferings, and great anguish of body.

Natrum Sulphuricum

Great sleepiness during day, especially in forenoon; failing asleep while reading or writing. Narcolepsy. Sleeplessness caused by great agitation. Uneasy sleep, with anxious and disagreeable dreams. Jerking of limbs during sleep. Starting as if in a fright, soon after falling asleep. Twitching of extremities during sleep – more so after midnight. Trembling of hands-on waking, and also when writing.

Nux Vomica

Goes to sleep late from crowding of thoughts on him. Great disposition to sleep, principally when rising in morning, or after dinner, or early in evening, and often with sleeplessness at night. Narcolepsy. Gentle and prolonged sleep-in morning, with difficult waking. Sleep too short, with difficulty in going to sleep again before midnight. Flow of ideas in bed in evening. Yawning and sleepiness during day, yawning with stretching of limbs. Sleeps mostly lying on the back. Loud snoring respiration during sleep. Comatose state, with heavy and profound sleep during day. Light nocturnal sleep, with frequent waking, or like a kind of coma vigil, with reveries full of troubles and agitation, and a sort of weariness as if the night were too long.-Sleeplessness from flatus. During sleep: frequent starts with fright, groans, lamentations, much talking, weeping; delirium, with an impulsive desire to run away from the bed, stertorous or whistling respiration, the patient lying on his back, with the arms raised over the head.

Opium

Lethargy, with snoring and mouth open, eyes open and convulsed, face red, and puffed, jaw hanging, loss of consciousness, difficult, slow, or intermittent respiration, pulse slow, or even suppressed, and convulsive movement of muscles of face, corners of mouth, and limbs. narcolepsy. Narcolepsy, urgent inclination to sleep, with absolute inability to go to sleep. Coma vigil. Incomplete sleep, without power to wake. Uneasy sleep, with anxious dreams. Stupefying, unrefreshing sleep. During sleep, picking of bedclothes; groaning, moaning (whining), terrific shocks in limbs. Nightmare. Lascivious, frightful, and anxious dreams. Dreams and cannot be roused. Pleasant, fantastic dreams.

Physostigma Venenosum

Uncommon mental activity. Foolish actions said it made him crazy. Exhilarated in morning, gloomy towards noon. Nothing was right, too many things in room; continually counting them. Irritable. Nervous, cannot endure pain. Exhaustion cannot remember anything. Disinclined to work. Difficult thinking; cannot concentrate the mind. Narcolepsy: Irresistible desire to sleep; soporific sleep. Restless sleep with dreams. During sleep mind so active was unaware he had been asleep. Falls asleep frequently but awakes suddenly without relief; following night scarcely closed eyes in sleep; thoughts very active; an idea once started kept on with unusual persistence.

Pulsatilla Nigricans

Irresistible sleepiness in afternoon. Great sleepiness during day, wakes confused, languid, unrefreshed. Narcolepsy. Sleepy while eating. Wide awake in the evening, first sleep restless. Lies with hands over head or crossed on abdomen and feet drawn up. Chattering in sleep. Talks, whines, or screams during sleep. Involuntary laughter and weeping. Great anguish and inquietude, mostly in precordial region, sometimes with inclination to commit suicide.

Sabadilla

Great inclination to sleep during day. Comes on when thinking, meditating, or reading. Agitated and unrefreshing sleep at night with anxious dreams. In the morning he starts up from his sleep as from a fright.

Zincum Metallicum

Narcolepsy. Sleep by day, and continued disposition to sleep, esp. in morning or after a meal; with inclination to yawn. Unconquerable drowsiness. Retarded sleep. Disturbed sleep, with frequent waking. Unrefreshing sleep with unpleasant dreams. Fantastic, frightful, agitated, or disgusting and terrific dreams, with talking and cries during sleep. Shocks in body during sleep and frequent starts.

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P. S: This article is only for doctors having good knowledge about Homeopathy and allopathy, for learning purpose(s).

For proper consultation and treatment, please visit our clinic.

Location, address and contact numbers are given below.

None of above-mentioned medicine(s) is/are the full/complete treatment, but just hints for treatment; every patient has his/her own constitutional medicine.

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Dr. Sayyad Qaisar Ahmed (MD {Ukraine}, DHMS), Abdominal Surgeries, Oncological surgeries, Gastroenterologist, Specialist Homeopathic Medicines.

  Senior research officer at Dnepropetrovsk state medical academy Ukraine.

Location:  Al-Haytham clinic, Umer Farooq Chowk Risalpur Sadder (0923631023, 03119884588), K.P.K, Pakistan.

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