Lupus Systemic Lupus Erythematosus or SLE.
Thank you for reading this post, don't forget to subscribe!Systemic lupus erythematosus (SLE) is a chronic autoimmune disease and is characterized by the production of unusual antibodies in the blood.
What is systemic lupus erythematosus? What are the different types of lupus?
Lupus is an autoimmune disease characterized by acute and chronic inflammation of various tissues of the body. Autoimmune diseases are illnesses that occur when the body’s tissues are attacked by its own immune system. The immune system is a complex system within the body that is designed to fight infectious agents, such as bacteria and other foreign microbes. One of the ways that the immune system fights infections is by producing antibodies that bind to the microbes. People with lupus produce abnormal antibodies in their blood that target tissues within their own body rather than foreign infectious agents. These antibodies are referred to as autoantibodies.
Because the antibodies and accompanying cells of inflammation can affect tissues anywhere in the body, lupus has the potential to affect a variety of areas. Sometimes lupus can cause disease of the skin, heart, lungs, kidneys, joints, and/or nervous system.
- When only the skin is involved by rash, the condition is called lupus dermatitis or cutaneous lupus erythematosus.
- A form of cutaneous lupus erythematosus that can be isolated to the skin, without internal disease, is called discoid lupus erythematosus.
- When internal organs are involved, the condition is referred to as systemic lupus erythematosus (SLE).
Both discoid lupus erythematosus and systemic lupus are more common in women than men (about eight times more common). The disease can affect all ages but most commonly begins from 20-45 years of age.
The cause(s) of systemic lupus erythematosus is (are) unknown, however, heredity, viruses, ultraviolet light, and drugs all may play some role.
I think that the root cause of lupus (SLE) could be side effects of some drugs, mineral unbalance in organism especially du to liver malfunctioning and/or liver diseases, said Dr Qaisar Ahmed.
Lupus signs and symptoms
-
- Butterfly-shaped rash on the face,
- appetite loss,
- joint pain,
- fever,
- photosensitivity,
- pleuritis,
- pericarditis,
- fatigue or feeling tired,
- muscle aches,
- hair loss (alopecia),
- arthritis,
- ulcers of the mouth and nose,
- chest pain caused by inflammation of the lining that surrounds the lungs (pleuritis) and the heart (pericarditis), and
- poor circulation to the fingers and toes with cold exposure (Raynaud’s phenomenon).
Up to 10% of people with lupus isolated to the skin will develop the systemic form of lupus (SLE).
Allopathic treatment of systemic lupus erythematosus is directed toward decreasing inflammation and/or the level of autoimmune activity with anti-inflammatory medications for those with mild symptoms and corticosteroids and/or medications that suppress the immune system for those with more severe lupus.
People with systemic lupus erythematosus can prevent “flares” of disease by avoiding sun exposure, not abruptly discontinuing medications, and monitoring their condition with their doctor.
More specific symptoms include skin changes, ulcers of the mouth and nose, photosensitivity (sensitivity to sunlight), and decreased circulation to the fingers and toes with cold exposure.
What are risk factors and causes of Systemic Lupus Erythematosus? Is lupus hereditary? Is lupus contagious?
Its confirmed that Lupus is not caused by an infectious microorganism and is not contagious from one person to another.
Genetic factors increase the tendency of developing autoimmune diseases, and autoimmune diseases such as lupus, rheumatoid arthritis, and autoimmune thyroid disorders are more common among relatives of people with lupus than in the general population.
It is possible to have more than one autoimmune disease in the same individual. Therefore, “overlap” syndromes of lupus and rheumatoid arthritis, or lupus and scleroderma, etc., can occur.
Some women with systemic lupus erythematosus can experience worsening of their symptoms before their menstrual periods. This phenomenon, together with the female predominance of systemic lupus erythematosus, suggests that female hormones play an important role in the expression of SLE. This hormonal relationship is an active area of ongoing study by scientists.
Research has demonstrated evidence that a key enzyme’s failure to dispose of dying cells may contribute to the development of systemic lupus erythematosus. The enzyme, DNase1, normally eliminates what is called “garbage DNA” and other cellular debris by chopping them into tiny fragments for easier disposal. Researchers turned off the DNase1 gene in mice. The mice appeared healthy at birth, but after six to eight months, the majority of mice without DNase1 showed signs of systemic lupus erythematosus. Thus, a genetic mutation in a gene that could disrupt the body’s cellular waste disposal may be involved in the initiation of systemic lupus erythematosus.
What is allopathic drug-induced Systemic Lupus Erythematosus?
Dozens of medications have been reported to trigger SLE. However, more than 90% of cases of “drug-induced lupus” occurs as a side effect of one of the following six drugs:
- hydralazine is used for high blood pressure;
- quinidine and procainamide are used for abnormal heart rhythms;
- phenytoin is used for epilepsy;
- isoniazid (Nydrazid, Laniazid) is used for tuberculosis; and
- d-penicillamine (used for rheumatoid arthritis).
These allopathic drugs are known to stimulate the immune system and cause SLE.
Fortunately, allopathic drug-induced SLE is infrequent (accounting for less than 5% of all patients with SLE) and usually resolves when the medications are discontinued.
Complications
Complications of organ involvement can lead to further symptoms that depend on the organ affected and the severity of the disease.
SLE-associated skin manifestations can sometimes lead to scarring. In discoid lupus, only the skin is typically involved. The skin rash in discoid lupus often is found on the face and scalp. It usually is red and may have raised borders. Discoid lupus rashes are usually painless and do not itch, but scarring can cause permanent hair loss (alopecia); Often, this occurs simultaneously with an increase in the activity of their disease. The hair loss can be patchy or diffuse and appear to be more like hair thinning. Over time, 5%-10% of those with discoid lupus may develop SLE.
Over half of the patients with SLE develop a characteristic red, flat facial rash over the bridge of their nose. Because of its shape, it is frequently referred to as the “butterfly rash” of SLE. The rash is painless and does not itch. The facial rash, along with inflammation in other organs, can be precipitated or worsened by exposure to sunlight, a condition called photosensitivity. This photosensitivity can be accompanied by worsening inflammation throughout the body, called a “flare” of the disease.
Typically, with treatment, this rash can heal without permanent scarring.
Joints etc
Most SLE patients will develop arthritis during the course of their illness. Arthritis from SLE commonly involves swelling, pain, stiffness, and even deformity of the small joints of the hands, wrists, and feet. Sometimes, the arthritis of SLE can mimic that of rheumatoid arthritis (another autoimmune disease).
Internal organs
More serious organ involvement with inflammation occurs in the brain, liver, and kidneys. White blood cells can be decreased in SLE (referred to as leukopenia or leucopenia). Also, a decrease in the number of blood-clotting factors called platelets (thrombocytopenia) can be caused by lupus. Leukopenia can increase the risk of infection, and thrombocytopenia can increase the risk of bleeding. Low red blood cell counts (hemolytic anemia) can occur.
Muscles and Vessels
Inflammation of muscles (myositis) can cause muscle pain and weakness. This can lead to elevations of muscle enzyme levels in the blood.
Inflammation of blood vessels (vasculitis) that supply oxygen to tissues can cause isolated injury to a nerve, the skin, or an internal organ. The blood vessels are composed of arteries that pass oxygen-rich blood to the tissues of the body and veins that return oxygen-depleted blood from the tissues to the lungs. Vasculitis is characterized by inflammation with damage to the walls of various blood vessels. The damage blocks the circulation of blood through the vessels and can cause injury to the tissues that are supplied with oxygen by these vessels.
Respiratory
Inflammation of the lining of the lungs (pleuritis) with pain aggravated by deep breathing (pleurisy) and of the heart (pericarditis) can cause sharp chest pain. The chest pain is aggravated by coughing, deep breathing, and certain changes in body position. The heart muscle itself rarely can become inflamed (carditis). It has also been shown that young women with SLE have a significantly increased risk of heart attacks due to coronary artery disease.
Urinary
Kidney inflammation in SLE (lupus nephritis) can cause leakage of protein into the urine, fluid retention, high blood pressure, and even kidney failure. This can lead to further fatigue and swelling (edema) of the legs and feet. With kidney failure, machines are needed to cleanse the blood of accumulated waste products in a process called dialysis.
Central and Peripheral Nervous system
Involvement of the brain can cause personality changes, thought disorders (psychosis), seizures, and even coma. Lupus of the nervous system (neurologic lupus) can lead to damage to nerves causing numbness, tingling, and weakness of the involved body parts or extremities. Brain involvement is referred to as lupus cerebritis.
Some people with SLE have Raynaud’s phenomenon. Raynaud’s phenomenon causes the blood vessels of the hands and feet to spasm, especially upon exposure to cold. The blood supply to the fingers and/or toes then becomes compromised, causing blanching, whitish and/or bluish discoloration, and pain and numbness in the exposed fingers and toes.
Other diseases and conditions that can accompany lupus include fibromyalgia, coronary heart disease, nonbacterial valvular heart disease, pancreatitis, esophagus disease with difficulty swallowing (dysphagia), swollen lymph nodes (lymphadenopathy), liver disease (lupoid hepatitis), infections, and a tendency to spontaneous blood clotting and thrombosis.
Diagnoses
Since SLE patients can have a wide variety of symptoms and different combinations of organ involvement, no single blood test establishes the diagnosis of systemic lupus.
To help doctors improve the accuracy of the diagnosis of SLE, 11 criteria were established by the doctors. These 11 criteria are closely related to the symptoms discussed above.
Some people suspected of having SLE may never develop enough criteria for a definite diagnosis. Other people accumulate enough criteria only after months or years of observation. When a person has four or more of these criteria, the diagnosis of SLE is strongly suggested.
Nevertheless, the diagnosis of SLE may be made in some settings in people with only a few of these classical criteria, and treatment may sometimes be instituted at this stage. Of these people with minimal criteria, some may later develop other criteria, but many never do.
The following are 11 criteria used for diagnosing systemic lupus erythematosus:
- Malar rash (over the cheeks of the face)
- Discoid skin rash (patchy redness with hyperpigmentation and hypopigmentation that can cause scarring)
- Photosensitivity (skin rash in reaction to sunlight [ultraviolet light] exposure)
- Mucous membrane ulcers (spontaneous sores or ulcers of the lining of the mouth, nose, or throat)
- Arthritis (two or more swollen, tender joints of the extremities)
- Pleuritis or pericarditis (inflammation of the lining tissue around the heart or lungs, usually associated with chest pain upon breathing or changes of body position)
- Kidney abnormalities (abnormal amounts of urine protein or clumps of cellular elements called casts detectable with a standard urinalysis)
Note: Ultimately, in patients with kidney disease from systemic lupus erythematosus (lupus nephritis), a kidney biopsy may be necessary to both define the cause of the kidney disease as being lupus-related as well as to determine the stage of the kidney disease in order to optimally guide treatments. Kidney biopsies are often performed by fine-needle aspiration of the kidney under radiology guidance, but in certain circumstances, a kidney biopsy can be done during an open abdominal operation. - Brain irritation (manifested by seizures [convulsions] and/or psychosis, referred to as “lupus cerebritis”)
- Blood-count abnormalities: low white blood count (WBC) or red blood count (RBC), or platelet count on routine complete blood count testing; leukopenia, anemia, and thrombocytopenia, respectively. Each of these is detectable with standard complete blood count testing (CBC).
- Immunologic disorder (abnormal immune tests include anti-double-stranded DNA (anti-dsDNA) or anti-Sm [anti-Smith] antibodies, falsely positive blood test for syphilis, anticardiolipin antibodies, lupus anticoagulant, or positive LE prep test)
- Antinuclear antibody (positive ANA antibody testing [antinuclear antibodies in the blood])
In addition to the 11 criteria, other tests can be helpful in evaluating people with SLE to determine the severity of organ involvement. These include routine testing of the blood to detect inflammation (for example, the erythrocyte sedimentation rate, or ESR, and the C-reactive protein, or CRP), blood-chemistry testing, direct analysis of internal body fluids, and tissue biopsies.
Abnormalities in body fluids (joint or cerebrospinal fluid) and tissue samples (kidney biopsy, skin biopsy, and nerve biopsy) can further support the diagnosis of SLE. The appropriate testing procedures are selected for the patient individually by the doctor.
What allopathic specialties treat Systemic Lupus Erythematosus?
Lupus is treated by internal medicine subspecialists called rheumatologists. Depending on whether or not specific organs are targeted, other health specialists who can be involved in the care of patients with lupus include
- dermatologists,
- nephrologists,
- hematologists,
- cardiologists,
- pulmonologist, and
- neurologists.
It’s not uncommon that a team of such physicians is coordinated by the treating rheumatologist together with the primary care doctor.
What Homeopathic specialties treat Systemic Lupus Erythematosus?
In Homeopathy lupus is not uncurbable or threatening disease and is easily curable, any Homeopathic doctor can easily handle even serious and chronic cases of lupus.
What is the allopathic treatment for Systemic Lupus Erythematosus?
There is no allopathic permanent cure for SLE. The allopathic goal of treatment is to relieve symptoms and protect organs by decreasing inflammation and/or the level of autoimmune activity in the body.
The precise treatment is decided on an individual basis. Many patients with mild symptoms may need no treatment or only intermittent courses of anti-inflammatory medications. Those with a more serious illness involving damage to internal organ(s) may require high doses of corticosteroids in combination with other allopathic drugs that suppress the body’s immune system.
To protect from sun sensitivity, sunscreens, sun avoidance, and sun protection clothing are used. Certain types of lupus rash can respond to topical cortisone medications.
Nonsteroidal anti-inflammatory drugs (NSAIDs) help reduce inflammation and pain in muscles, joints, and other tissues. Examples of NSAIDs include aspirin, ibuprofen, naproxen, and sulindac.
Since the individual response to NSAIDs varies, it is common for an allopathic doctor to try different NSAIDs to find the most effective one with the fewest side effects.
Corticosteroids are more potent than NSAIDs in reducing inflammation and restoring function when the disease is active. Corticosteroids are particularly helpful when internal organs are affected. Corticosteroids can be given by mouth, injected directly into the joints and other tissues, or administered intravenously.
Hydroxychloroquine is an antimalarial medication found to be particularly effective for SLE patients with fatigue, skin involvement, and joint disease. Consistently taking hydroxychloroquine can prevent flare-ups of lupus. Hydroxychloroquine is commonly used in combination with other treatments for lupus.
For resistant skin disease, other antimalarial drugs, such as chloroquine (Aralen) or quinacrine, are considered and can be used in combination with hydroxychloroquine. Alternative medications for skin disease include dapsone and retinoic acid (Retin-A). Retin-A is often effective for an uncommon wart-like form of lupus skin disease.
Medications that suppress immunity (immunosuppressive medications) are also called cytotoxic drugs. They are sometimes referred to as chemotherapy because they are also used to treat cancer, generally in much higher doses than those used to treat lupus. Immunosuppressive medications are used for treating people with more severe manifestations of SLE, such as damage to internal organs.
All immunosuppressive medications can seriously depress blood-cell counts and increase the risk of infection and bleeding. Immunosuppressive medications may not be taken during pregnancy or conception because of the risk to the fetus. Other side effects are specific to each drug. For example, methotrexate can cause liver toxicity, while cyclosporine can impair kidney function.
Another immunosuppressant, mycophenolate mofetil has been used as an effective medication for lupus, particularly when it is associated with kidney disease. CellCept has helped reverse active lupus kidney disease (lupus renal disease) and in maintaining remission after it is established. Its lower side-effect profile has an advantage over traditional immune-suppression medications.
In lupus patients with serious brain (lupus cerebritis) or kidney disease (lupus nephritis), plasmapheresis is sometimes used to remove antibodies and other immune substances from the blood to suppress immunity. Plasmapheresis is a process of removing blood and passing the blood through a filtering machine, then returning the blood to the body with its antibodies removed. Serious lupus nephritis is treated aggressively because it can progress to end-stage kidney disease. End-stage kidney damage from SLE requires dialysis and/or a kidney transplant.
Rarely, people with SLE can develop seriously low platelet levels, thereby increasing the risk of excessive and spontaneous bleeding. Since the spleen is believed to be the major site of platelet destruction, surgical removal of the spleen is sometimes performed to improve platelet levels. Other treatments have included plasmapheresis and the use of male hormones.
Plasmapheresis has also been used to remove certain harmful proteins (cryoglobulins) that can lead to vasculitis (inflammation of the blood vessels, which can cause damage to organs).
Allopathic research is indicating the benefits of rituximab in treating lupus. Rituximab is an intravenously infused antibody that suppresses a particular white blood cell, the B cell, by decreasing its number in the circulation. B cells have been found to play a central role in lupus activity, and when they are suppressed, the disease tends toward remission. This may be particularly helpful for people with kidney disease.
Another B-cell-suppressing treatment is belimumab. Belimumab blocks the stimulation of the B cells (a B-lymphocyte stimulator or BLyS-specific inhibitor) and is approved by allopathic researchers for the treatment of adults with active autoantibody-positive systemic lupus erythematosus who are receiving standard therapy.
Saphnelo is indicated for the treatment of adult patients with moderate to severe systemic lupus erythematosus (SLE) who are receiving standard therapy. The medication is given intravenously, every 4 weeks. It is not indicated for severe active central nervous system lupus (such as cerebritis) or severe active lupus nephritis (serious kidney inflammation caused by lupus). Saphnelo medication works differently than other lupus medications as it blocks type 1 interferon (IFN-1) activity. IFN-1 is elevated (upregulated) in SLE. By blocking the IFN-1 receptor, lupus activity is likely to improve, and it may be possible to decrease the use of steroid medication (prednisone). Saphnelo may work best for arthritis caused by lupus and lupus skin rashes. Because Saphnelo is an immune-suppressing medication, there is an increased risk of infections and serious infections when taking the medication. Some patients had severe allergic reactions (anaphylaxis) in the studies. Patients being treated with Saphnelo should not receive live vaccines while under treatment with the medication.
Side effects of Allopathic drugs
The most common side effects are stomach upset, abdominal pain, ulcers, and even ulcer bleeding. NSAIDs are usually taken with food to reduce side effects. Sometimes, medications that prevent ulcers while taking NSAIDs, such as misoprostol, are given simultaneously.
Corticosteroids have serious side effects when given in high doses or over prolonged periods, and the doctor will try to monitor the activity of the disease to use the lowest doses that are safe.
Side effects of corticosteroids include weight gain, thinning of the bones and skin, infection, diabetes, facial puffiness, cataracts, and death (necrosis) of the tissues in large joints (called avascular necrosis or AVN).
Side effects are uncommon but include diarrhea, upset stomach, and eye-pigment changes. Eye-pigment changes are rare but require monitoring by an ophthalmologist (eye specialist) during treatment with Hydroxychloroquine.
Researchers have found that Hydroxychloroquine significantly decreased the frequency of abnormal blood clots in people with systemic lupus.
This fascinating study highlights an important reason for patients and doctors to consider Hydroxychloroquine for long-term use, especially for those SLE patients who are at some risk for blood clots in veins and arteries, such as those with phospholipid antibodies (cardiolipin antibodies, lupus anticoagulant, and false-positive venereal disease research laboratory test).
Is there a Systemic Lupus Erythematosus diet?
It is generally recommended that patients with lupus eat a balanced diet that includes plant-based foods and lean sources of protein. It is very important for people with lupus to eat a healthy diet because it helps the body to function optimally.
According to allopathic doctors DHEA (dehydroepiandrosterone) has been helpful in reducing fatigue, improving thinking difficulties, and improving the quality of life; But recent research indicates that DHEA diet supplementation has no any good results.
How can systemic lupus erythematosus affect pregnancy or the newborn?
Pregnant women with SLE are considered high-risk pregnancies. Women with SLE who are pregnant require close observation during pregnancy, delivery, and the postpartum period. This includes fetal monitoring by the obstetrician during later pregnancy. These women can have an increased risk of miscarriages (spontaneous abortions) and can have flares of SLE during pregnancy (due to the presence of phospholipid antibodies, such as cardiolipin antibodies or lupus anticoagulants, in the blood, Cardiolipin antibodies are associated with a tendency toward blood clotting).
That’s women with SLE who have cardiolipin antibodies or lupus anticoagulants may need blood-thinning medications (aspirin with or without low molecular weight heparin) to prevent miscarriages.
Other reported treatments include the use of intravenous gamma globulin for selected patients with histories of premature miscarriage and those with low blood-clotting elements (platelets) during pregnancy.
Some allopathic doctors thinks that corticosteroids, such as prednisone, are also safely used to treat certain manifestations of lupus during pregnancy; But I (Dr Qaisar Ahmed) strongly oppose it.
Lupus antibodies can be transferred from the mother to the fetus and result in lupus illness in the newborn (“neonatal lupus”). This includes the development of low red cell counts (hemolytic anemia) and/or white blood cell counts (leucopenia) and platelet counts (thrombocytopenia) and skin rash. Problems can also develop in the electrical system of the baby’s heart (congenital heart block). Occasionally, a pacemaker for the baby’s heart is needed in this setting.
Homeopathic Treatment for Systemic Lupus Erythematosus
Arsenicum Album
Debility, exhaustion, and restlessness, with nightly aggravation. Circumscribed flush of cheeks. Itching, burning, swellings; oedema, eruption, papular, dry, rough, scaly; worse cold and scratching. Malignant pustules. Ulcers with offensive discharge. Anthrax. Poisoned wounds. Urticaria, with burning and restlessness. Psoriasis. Scirrhus. Icy coldness of body. Epithelioma of the skin. Gangrenous inflammations.
High temperature. Periodicity marked with adynamia. Septic fevers. Intermittent. Paroxysms incomplete, with marked exhaustion.
Mezereum
Skin symptoms, affections of bones, and neuralgias most important, especially about teeth and face. Eruptions after vaccination. Burning, darting sensation in the muscles. Face red,eruption around mouth, sometimes with coryza. Eczema; intolerable itching; chilliness with pruritus; worse in bed. Ulcers itch and burn, surrounded by vesicles and shining, fiery-red areola. Zona, with burning pain.
Bones, especially long bones, inflamed and swollen; caries, exostosis; pain worse night, touch, damp weather. Eruptions ulcerate and form thick scabs under purulent matter exudes.
Rhus Toxicodendron
Face: Jaws crack when chewing. Easy dislocation of jaw (Ign; Petrol). Swollen face, erysipelas. Cheek bones sensitive to touch. Parotitis. Facial neuralgia, with chilliness; worse, evening. Crusta lactea.
Skin red, swollen; itching intense. Vesicles, herpes; urticaria; pemphigus; erysipelas; vesicular suppurative forms. Glands swollen. Cellulitis. Burning eczematous eruptions with tendency to scale formation.
Apis Melifestida
Skin swellings after; sore, sensitive. Stinging. Erysipelas, with sensitiveness and swelling, rosy hue. Carbuncles, with burning, stinging pain (Ars; Anthrac). Sudden puffing up of whole body.
Natrum Muriaticum
Greasy, oily, especially on hairy parts. Dry eruptions. Fever blisters. Urticaria; itch and burn. Crusty eruptions in bends of limbs, margin of scalp, behind ears (Caust). Warts on palms of hands. Eczema; raw, red, and inflamed; worse, eating salt, at seashore. Affects hair follicles. Alopecia. Hives, itching after exertion.
Belladonna
Skin: Dry and hot; swollen, sensitive; burns scarlet, smooth. Eruption like scarlatina, suddenly spreading. Erythema; pustules on face. Glands swollen, tender, red. Boils. Acne rosacea. Suppurative wounds. Alternate redness and paleness of the skin. Indurations after inflammations. Erysipelas.
Face: Red, bluish-red, hot, swollen, shining; convulsive motion of muscles of face. Swelling of upper lip. Facial neuralgia with twitching muscles and flushed face.
Bellis Perennis
Boils. Ecchymosis, swelling, very sensitive to touch. Venous congestion due to mechanical causes. Varicose veins with bruised sore feeling. Exudations and swellings. Acne.
Tarentula Hispanica
Ecchymosed spots. Hepatic spots. Furfuraceous spots. Miliary eruption. Indolent pimples. Vesicular eruption like crusta lactea. A small callosity, whitish, indolent, between middle and index fingers, increased, with heat and pain, extended, broke, leaving an ulcer with callus edges, healed leaving a small scar. Painful callosity at end of thumb fell off. Callosity in index fell out. Every year intense pain in toes from reopened wound. Formication; pricking; itching over whole body.
Kreosotum
Skin soft, unnatural feel of skin, with pegged teeth. Violent itching all over body, especially towards evening, and with burning sensation after scratching. Nettle-rash.-Eruption, like bug-bites, with violent itching. Large, greasy-looking, pox-shaped pustules. Mealy and pustular, dry or humid tetters with violent itching.
Frequent, and even constant heat in face, with throbbing in cheeks and forehead, and with a deep red colour of whole face. Acne. Face pale green with swelling of cervical glands. Greyish, earthy colour of the face. Furfuraceous tetters on cheeks, on eyelids, and round mouth. Acute drawing pain.
Sulphur
Dry, scaly, unhealthy skin; every little injury suppurates. Freckles. Itching, burning; worse scratching and washing. Pimply eruption, pustules, rhagades, hangnails. Excoriation. Feeling of a band around bones. Skin affections after local medication. Pruritus, Herpes across the nose. Nose stuffed indoors. Imaginary foul smells. Alae red and scabby. Chronic dry catarrh; dry scabs and readily bleeding. Polypus and adenoids.
Dulcamara
Adenitis. Pruritus, always worse in cold, wet weather. Herpes zoster, pemphigus. Swelling and indurated glands from cold. Vesicular eruptions. Sensitive bleeding ulcers. Little boils. Red spots, urticaria, brought on by exposure, or sour stomach. Humid eruptions on face, genitals, hands, etc. Warts, large, smooth, on face and palmar surface of hands. Anasarca. Thick, brown-yellow crusts, bleeding when scratched. Humid eruption on cheeks and face. Feels as if cobwebs on face. Eczema of nose. Itching pimples. Moist eczema around mouth and chin. Erysipelas, burning and stinging.
Thyroidinum
Psoriasis associated with adiposity (not in developing stage). Skin dry, impoverished. Cold hands and feet. Eczema. Uterine fibroids. Browne swelling. Swelling of glands of stony hardness. Sluggish cases. Jaundice with pruritus. Ichthyosis, lupus. Itching without eruption, worse night.
Sepia Officinalis
Herpes circinatus in isolated spots. Itching; not relieved by scratching; worse in bends of elbows and knees. Chloasma; herpetic eruption on lips, about mouth and nose. Ringworm-like eruption every spring. Urticaria on going in open air; better in warm room. Hyperhidrosis and bromhidrosis. Sweat on feet, worse on toes; intolerable odor. Lentigo in young women. Ichthyosis with offensive odor of skin. Rosacea; saddle-like brownish distribution on nose and cheeks.
Ranunculus Bulbosus
Burning and intense itching; worse, contact. Hard excrescences. Herptic eruptions, with great itching. Shingles, bluish vesicles. Itching in palms. Blister-like eruption in palms. Corns sensitive. Horny skin. Fingertips and palms chapped. Vesicular and pustular eruptions.
Psorinum
Dirty, dingy look. Dry, lusterless, rough hair. Intolerable itching. Herpetic eruptions, especially on scalp and bends of joints with itching; worse, from warmth of bed. Enlarged glands. Sebaceous glands secrete excessively; oily skin. Indolent ulcers, slow to heal. Eczema behind ears. Crusty eruptions all over. Urticaria after every exertion. Pustules near fingernails. Humid eruption on face.
Phytolacca Decandra
Itches, becomes dry, shrunken, pale. Papular and pustular lesions. Most useful in early stages of cutaneous diseases. Disposition to boils, and when sloughing occurs. Squamous eruptions. Syphilitic eruptions. Swelling and induration of glands. Venereal buboes. Scarlatina-like rash. Warts and moles.
Iris Versicolor
Herpes zoster, associated with gastric derangements. Pustular eruptions. Psoriasis; irregular patches with shining scales. Eczema, with nightly itching.
Acid Phosphoricum
Pimples, acne, blood-boils. Ulcers, with very offensive pus. Burning red rash. Formication in various parts. Falling out of the hair. Tendency to abscess after fevers. On face feeling of tension as from dried albumen.
Silice Tera
Felons, abscesses, boils, old fistulous ulcers. Delicate, pale, waxy. Cracks at end of fingers. Painless swelling of glands. Rose-colored blotches. Scars suddenly become painful. Pus offensive. Promotes expulsion of foreign bodies from tissues. Every little injury suppurates. Long lasting suppuration and fistulous tracts. Dry finger tips. Eruptions itch only in daytime and evening. Crippled nails. Indurated tumors. Abscesses of joints. After impure vaccination. Bursa. Lepra, nodes, and coppery spots. Keloid growths. Eruption on chin. Facial neuralgia, throbbing, tearing, face red.
Acid Muriaticum
Papular and vesicular eruptions, with great itching. Carbuncles; foul-smelling ulcers on lower extr Psoriasis. Erysipelas in face. Itching on back of finger-joints. Unhealthy skin; slight injuries suppurate. Herpes (Rhus). Erysipelatous inflammation with swelling and tension. Chilblains relieved in open air. Trade eruptions on fingers and hands, itching and stinging. Ends of hair become tangled.emities. Scarlet fever, livid, with petechiae; scanty eruption. Eczema on back of hands. Pimples and freckles; lips raw, dry, cracked.
Borax
Psoriasis. Erysipelas in face. Itching on back of finger-joints. Unhealthy skin; slight injuries suppurate. Herpes. Erysipelatous inflammation with swelling and tension. Chilblains relieved in open air. Trade eruptions on fingers and hands, itching and stinging. Ends of hair become tangled. Face swollen, with pimples on nose and lips. Feeling of cobwebs.
Antimonium Crudum
Eczema with gastric derangements. Pimples, vesicles, and pustules. Sensitive to cold bathing. Thick, hard, honey-colored scabs. Urticaria; measle-like eruption. Itching when warm in bed. Dry skin. Warts. Dry gangrene. Scaly, pustular eruption with burning and itching. Pimples, pustules, and boils on face. Yellow crusted eruption on cheeks and chin. Sallow and haggard.
For consultation about Systemic Lupus Erythematosus, Psoriatic and its Treatment, Diet etc. visit our clinic.
To order medicine by courier, please send your details at WhatsApp– +92319884588
Dr. Sayyad Qaisar Ahmed (MD {Ukraine}, DHMS) ; senior research officer Dnepropetrovsk state medical academy Ukraine; is a leading Homeopathic physician practicing in Al-Haytham clinic, Umer Farooq Chowk Risalpur Sadder (0923631023, 03119884588), K.P.K, Pakistan.
Like, subscribe and find more about Dr. Sayyad Qaisar Ahmed at :
https://www.youtube.com/Dr Qaisar Ahmed